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Muscles are essential to keeping the body healthy and functioning well. In addition to allowing us to move, they help regulate blood sugar, support metabolic health, and protect our joints while helping us remain independent. Yet, as we grow older, muscle strength and size naturally decline. This gradual loss can affect stability, raise the risk of falls, and make it take longer to bounce back after an injury or illness.
Scientists have identified a previously unknown mechanism that appears to influence the ability of older muscles to recover and repair damage. Essentially, exercise helps keep the muscles’ internal repair system working properly, allowing them to maintain healthy tissue and recover from damage.
The discovery centers on a gene that becomes overactive as we age, making it harder for muscles to clear damaged proteins and recover properly. Regular exercise helps restore this balance, encouraging muscles to repair themselves and rebuild strength. But when this system becomes too disrupted, the positive effects of exercise may be reduced.
To investigate the gene's effects, scientists conducted experiments in fruit flies and mice. In both species, muscle function declined when the gene became more active and improved when its activity was reduced. The fact that the results were consistent across two different animals adds support to the findings.
By better understanding this process and learning how to regulate the gene, scientists may be able to recreate some of the beneficial changes that exercise triggers in muscles. This knowledge could eventually help researchers find new ways to protect and maintain muscle strength and mobility in older adults when physical activity is difficult or limited.
To view the original scientific study click below:
Exercise suppresses DEAF1 to normalize mTORC1 activity and reverse muscle aging/a>
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